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A highly selective inhibitor of inducible Nitric Oxide Synthase (iNOS).Attached is a representative reference from researchers who have used our 1400W Upregulation of Inducible Nitric Oxide Synthase Contributes to Attenuated Cutaneous Vasodilation in Essential Hypertensive Lysozyme, a mediator of sepsis, impairs the cardiac neural adrenergic response by nonendothelial release of NO and inhibitory G protein Depletion of Endogenous Nitric Oxide Enhances Cisplatin-induced Apoptosis in a p53-dependent Manner in Melanoma Cell Lines.Dextran sodium sulfate-induced colitis reveals nicotinic modulation of ion transport via iNOS-derived NO
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Lysozyme, a mediator of sepsis, impairs the cardiac neural adrenergic response by nonendothelial release of NO and inhibitory G protein signaling Steven N. Mink, Zhao-Qin Cheng, Ratna Bose, Hans Jacobs, Krika Kasian, Diane E. Roberts, Luis E. Santos-Martinez, and R. Bruce Light Am J Physiol Heart Circ Physiol, Nov 2007; 293: H3140 - H3149.
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